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And locking down is totally unnecessary, as it is already demonstrated it did zip so far anyway. Just a crazy world led by charlatans and their minions and supported by uneducated and scared people.Edit: As some uneducated misinformed specimen started to comment here, I want to addthat “Side-Effects” do not always manifest instantly. There are so many examples of long term effects from so many medicines and treatments which were not tested enough in time. There were tragic occurances happening even after 10 years, so do not sell me ignorance here.

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As we can see, the I_m not just daddy_s little girl i_m a Veteran_s daughter shirt moreover I love this variant is capable of establishing dominance very quickly over any pre-existing variants. In India, it went from ~10% of the cases to current ~80% in a matter of a month. In the UK, it went from almost negligible levels to almost 50% of the cases in a month. What is also very significant about its success in the UK is that it happens in the environment where the dominant virus species is the Kent variant (B.1.1.7), which spread very quickly through the UK, Europe, and most of the world due to its 50%-70% advantage in transmissibility. The current UK virus prevention and vaccination measures are keeping B.1.1.7 numbers down, yet B.1.617.2 has no problems growing in numbers. The linear slope of the plot indicates that the ratio of the two variants is changing exponentially over time, which is what you would expect when two variants with different transmissibilities are evolving within the same population. The slope of the line depends on the ratio of the transmissibilities of the two variants, which is nearly identical in all tree locations. The most accurate data comes from the UK (the green curve) due to the larger number of samples, and it produces the best defined value for the B.1617.2 transmissivity advantage over B.1.1.7: a factor of 2.26 (range 2.21–2.31), assuming a replication period of 5.5 days. This is how much the reproduction number (R0) increases from B.1.1.7 to B.1.617.2. This is also equivalent to saying that B.1.617.2 is gaining in numbers over B.1.1.7 by a factor of ~2.8 every week. This technique for calculating relative transmissibility has to be applied with caution, however, and could sometimes produce erroneous estimates. Ideally, we want both species to circulate within the same population group, to ensure that they both exist in the population with the same level of immunity and social interaction. When we only have the number of overall counts for the whole country to work with, there is no way to ensure that. In fact, in the case of the UK data, most of the B.1.617.2 cases come from the Bolton area, while the cases of B.1.1.7 are scattered across the whole country. So the data could be skewed to erroneously high values for B.1.617.2 if the Bolton area has much higher level of social interaction, or much lower level of population immunity than the rest of the country. The above charts show that up to approximately April 25, the case curve in Bolton resembled very closely the average case curve across the whole UK. Through most of April, the number of new cases (mainly B.1.1.7) remained relatively flat, and started taking off only when the Indian variant showed up in late April. In the first week of May, the number of cases increased by a factor of ~2.5, which is close to the weekly gain of B.1.617.2 over B.1.1.7 calculated above (2.8). All these factors seem to suggest that our estimate of B.1.617.2 transmissibility in the UK is pretty close to the actual value, and these numbers are also supported by New York and Japan data.